Congenital anomalies of the heart and vessels 10 Heart malformations are determined by various factors, some with severe movement disorders and oxygen that are incompatible with life, other compatible although initially not generally allow a long-term survival.
They occur hpv-positive squamous cell carcinoma of tonsil animals as lack or excess malformations by malformations of position, or structural alterations septs or the heart valves.
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Shows theoretical and practical importance: Acardia total lack of heartlack of closing the pericardial sac, diplocardia double heart multiplicitas cordis multiple cordsdextrocardia heart on the right side of the mediastinumcardiac ectopia presence of heart in the cervical region, pectoral or abdominaletc. Septs structural defects are common to all species.
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The total or partial lack thereof, determines the appearance of cords bilocular right or left respectively upper and lower or bilocular cords biventricular, trilocular, biatriale or malformations incompatible with life. Persistence of the oval hole foramen hpv-positive squamous cell carcinoma of tonsil persistence after birth is located in interarterial septum and favors the mixing of the arterial with venous blood eventually causing death by hypoxia.
In the structure of all valves, especially in the atrioventricular ones, can be found congenital hematic cyst with the diameter to an inch. Pericardium injuries Dystrophic lesions of the pericardium is of less importance in mammals, while in birds they produce contagion du papillomavirus changes sometimes incompatible with life.
Serous atrophy of the pericardium occurs in older animals, hpv-positive squamous cell carcinoma of tonsil the chronic wasting disease. Adipose tissue in the heart is replaced with a substantial amount of transudate.
The lesion is also known as gelatinous edema or "ex edema vacuo". Macroscopic instead of the fat tissue there is a yellow gelatinous mass-glassy, wet on section.
Serous atrophy has practical importance in veterinary checks for meat, it indicates a state of cachexia of the slaughtered animal.
Fat infiltration of epicardium is the opposite of serous atrophy. It is characterized by excessive storage of fat in this stage, and sometimes fat necrosis.
It is found mainly in hpv-positive squamous cell carcinoma of tonsil subjected to the process of fattening cattle, pigs, poultry. Melanosis pericardial is a congenital process which occurs in calves, lambs, etc. Pericardial Gout is found in birds, being the expression status of general uric diathesis.
Macroscopic, hpv-positive squamous cell carcinoma of tonsil the transparency of the pericardium is noticed in the pericardial cavity the presence of a white deposit like chalk more or less adherent to pericardium.
Epicardium appears sprinkled with white paint. Microscopic reveals acicular crystals arranged in rosettes or amorphous masses uropurinice. In longest developments on the outskirts of uric deposits a lymphohystocytar mesenchymal reaction is observed with the appearance of giant cells, ultimately resulting in foreign body granulomas granulomatous pericarditis.
Installing the inflammatory process is possible only in cases where the bird holding a long "uric poisoning", facilitating hpv-positive squamous cell carcinoma of tonsil emergence and development of granulomatous inflammation. Changes in blood and lymphatic circulation Pericardial bleeding, manifested by bruising and suffusions, characterize the poisoning, being the consequence of increased fragility of the capillary basement membrane. Macroscopically, on the surface of the pericardium and especially to the heart epicardium indicate the presence of small reddish points distributed over the deposit of fat and coronary sulcus.
Such lesions are found in diseases like: - bacterial septic emice: hpv-positive squamous cell carcinoma of tonsil, pasteurellosis, streptococci; - virotic: classical and African swine fever, Newcastle disease; - toxic: warfarin poisoning or with derivatives.
It is found in all species, but especially horses, pigs and dogs. Effusion is the accumulation of the transudation into the pericardial cavity. Pericardium and epicardium is shiny and smooth.
It occurs in all species, with greater practical importance in pigs and birds. Chylopericardium is the accumulation in the pericardial cavity of lymph as a result of breakagethe thoracic duct Macroscopic, the presence of a lipid-rich milky white liquid.
All "pericardial collections" described above, have a bad prognosis as may be the cause of sudden death in the so-called "cardiac tamponade".
Inflammation of the pericardium Inflammation of the pericardium are known under the generic name of pericarditis, it affects both blades of the pericardium and epicardium, the inflammatory process affects only the visceral foil.
Are commonly found in cattle, poultry, swine, horses and carnivores are produced by toxic agents, infections and trauma. From an evolutionary standpoint pericarditis may be acute, subacute and chronic.
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Pathologically are classified pericarditis: necrotic, serous, fibrinous, purulent, traumatic, Ihor, fibrotic and granulomatous. Necrotic pericarditis is common in lambs visceral necrobacillosis s being linked with lung necrotic foci. Macroscopic in the parietal serous pericardium are present outbreaks of gray-yellow, isolated or confluent, looking brittle, dry, of various sizes, surrounded by line bleeding in acute developments, less white in chronic developments. Serous Pericarditis is an inflammation observed in the evolution of acute septicaemic disease avian cholera, hydropericardium ruminants, etc.
Parietal and visceral pericardial blades are congested, shiny and wet.
Microscopic is observed vacuolation and desquamation of the mesothelial cells, congestion and edema subepicardic, leucodiapedesis. Serous pericarditis, stopped at this stage, has a benign evolution, being among the few sores that heal by "restitutio ad integrum". In general, it evolves into a serofibrinous inflammation and fibrinous eg avian choleraor the serohemorhagic inflammation eg coal emphysematous.
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Fibrinous pericarditis occurs in a number of infectious diseases and in the first phase of traumatic pericarditis in cattle. It is characterized by the appearance and accumulation of fibrinous exudate in the pericardial cavity.
Macroscopic, serous pericardium strips are opaque, thickened, rough, due to the deposition of fibrin on their surface. Pericardial cavity is enlarged and contains appreciable warts on hands boots of yellowish fibrin. Fibrinous exudate can unite the two skins of the serous pericardium, resulting in adhesions that can be easily removed.
In the fibrin mass can accumulate fibrin exudate serous or purulent or hemorrhagic.